Management of Uric Acid (Gouts Arthitis)

Uric acid is the acid form crystals that are the end product of purine metabolism (derived form nucleoprotein). Arthritis due to increased blood levels of uric acid or gout arthritis called arthritis arthritis. If not adequately treated gout can cause joint damage. Acute gouty arthritis is caused by inflammation of the tissue reaction to the formation of urate crystals. In the most cases of gout history of the disease and the clinical picture is specific, so sometimes the diagnosis can be directly enforced.Symptoms or clinical features often have a distinctive history of the disease, namely:- Acute Arthritis: Arthritis is a very heavy acute. Patients unable to walk (if that is affected by the foot), can not wear shoes, and sleep can be disturbed. Very severe pain. The pain reaches a peak within 24 hours after symptoms first started.Joint-Location: Acute attacks are usually monoartikular (strike only one joint) accompanied by symptoms of inflammation of the complete process, ie: red, swollen, palpable heat, and pain. The most frequent location of the first attack is the base of the big toe joint. Almost in all cases, the location of arthritis in peripheral joints and especially rare in the central joint.-Recovers perfectly between acute attacks. Acute attacks can be improved in the first attack, then followed by complete recovery without any symptoms until the next attack. If the state of hyperuricemia (elevated blood uric acid levels) are not corrected, there will be a chronic gouty arthritis. In the cured state outside of an acute attack the patient can operate normally as usual.-Hyperuricemia. Hyperuricemia is not always the same situation with acute gouty arthritis. This means that not always acute gout arthritis is accompanied by an increase in blood uric acid levels. Many people with elevated levels of blood uric acid, but never suffered an attack of gout or arthritis are TOFI. There are researchers who say that arthritis of gout and hyperuricemia are genetically determined by different genes. Rise and fall of blood uric acid levels may trigger an attack of acute gout arthritis.-TOFI, is the accumulation of urate crystals in tissues. Have distinctive properties as lumps under the skin of the nodes. TOFI most often arise in a person suffering from gout arthritis over 10 years. TOFI can occur without symptoms, for example in cartilage tissue in the ear. In the severe acute gouty arthritis TOFI can arise in the first such attack on the base of the thumb joint and thumb joint itself. With hyperuricemia TOFI uncontrolled, will increase in size that can cause changes in shape and impaired joint function.Arthritis, gout is not typical. This disease is not found on the symptoms of arthritis, monoartikular and complete recovery. TOFI are usually only found in patients who had been suffering from arthritis, gout can be found on the first attack. From the history of the disease do not get description pains, normal sleep and can run regular. In rare cases, can affect many joints, there is accumulation of urate in the muscles around the eye, the cornea and lens of the eye. Accumulation can also be obtained on the heart muscle, heart valve. Can also be found in the kidneys without primary attack of gout in the joints of arthritis symptoms.Management of gout arthritis:A. Relieves arthritis (with medication and rest the affected joint).2. Regulation of uric acid the body (with diet and medication).The main purpose of the treatment of gout arthritis are:a. Treating acute attacks as well and correctlyb. Prevent repeated attacks of acute gouty arthritisc. Prevent severe joint abnormalities due to accumulation of urate crystalsd. Prevent complications that can occur due to increased uric acid on the heart, kidneys and blood vessels.e. Prevent the formation of stones in the urinary tract.The sooner someone gets treatment from an acute attack, the sooner recovery.Treatment can be given anti-inflammatory drugs (antirheumatic) and uric acid-lowering drugs (drugs that speed up / increase spending through the urinary urate or drug that lowers uric acid production).Diabetes managementFoods should be avoided is that many foods contain high purine. High purine-containing foods (group A) is the liver, kidney, brain, heart, lungs, etc. offal, shrimp, mussels, clams, sardines, herring, meat extracts (shredded, beef jerky), yeast (tape), alcohol and food in cans. Foods that contain purines moderate (class B) is a fish that does not include class A, beef, dried beans, cauliflower, spinach, asparagus, beans, mushrooms, cassava leaves, papaya leaves, kale. Foods that contain purines lighter (class C) are cheese, milk, eggs, other vegetables, fruits. Diabetes management should be done when the uric acid levels exceed 7 mg / dl by not consuming food and class A limit themselves to mengonsmsi food group B. Also restrict ourselves to consume fat and are advised to drink plenty of water.

Source : Pikiran Rakyat Cyber Media